While marijuana, derived from the Cannabis sativa plant, is often perceived as harmless, emerging research suggests potential health risks, particularly for heart health. Smoking marijuana can rapidly introduce its effects, but it also brings many of the same toxins, irritants, and carcinogens found in cigarette smoke—a known contributor to heart disease and cancer.
Interestingly, the history of marijuana cultivation stretches back some 6,000 years, indicating its longstanding presence in human culture. However, the modern understanding of its impact, especially on heart health, is still developing. There are conditions such as cannabinoid hyperemesis syndrome (CHS), with symptoms akin to Crohn’s disease or gallbladder diseases, that frequent marijuana smokers may be at risk for.
Yet, it’s not all cautionary; studies have shown that cannabinoids like CBD can help reduce inflammation and neuropathic pain. Moreover, a 2018 study highlighted CBD’s potential in preventing relapse in drug and alcohol addiction, indicating a complex profile of benefits and risks.
The endocannabinoid system (ECS) plays a significant role in maintaining bodily homeostasis, yet its full implications are not entirely understood. With the ECS influencing an array of physiological processes, the impact of cannabis on this system is a critical area of study.
Harvard Medical School experts caution that while there are benefits of medical cannabis for some conditions, there are also non-benefits for others. Furthermore, risks become more pronounced for individuals over 55. The science behind cannabis use, especially in patients with serious illnesses, is still catching up with real-world applications. This gap highlights the necessity for more informed and engaged approaches to connect cannabis use with healthcare.
Long-term use of cannabis has been associated with an average decline of 5.5 points in IQ from childhood, along with deficits in learning and processing speed. The more frequent the use, the greater the cognitive impairment observed, suggesting a possible causative relationship.
Moreover, a comprehensive study by Stanford Medicine researchers indicates an increased risk of heart disease and heart attack among marijuana users. THC, the psychoactive component, is shown to cause inflammation in endothelial cells and atherosclerosis in mice, raising concerns about its cardiovascular effects in humans.
Amidst these findings, there remains an element of uncertainty regarding the full spectrum of marijuana’s effects on health. While some studies point to therapeutic benefits, the potential risks, particularly related to heart health, warrant a cautious approach.
